Publications
- The internalisation of chemokine receptor CXCR3 in the CD3+ cells after ligand binding could be the reason for increased Th1 synovial infiltration in rheumatoid arthritisPublication without faculty affiliation
- The role of CXCR3 internalisation after ligand binding in T cell infiltration of rheumatoid synoviumPublication without faculty affiliation
- Internalization of chemokine CXC receptor 3 in the CD3+ cells after ligand binding and endothelial transmigration suggests the mechanism for increased TH1 synovial infiltration in rheumatoid arthritisPublication without faculty affiliation
- Loss of surface CXCR3 expression in the RA synovial CD3+ cells as a result of ligand binding suggests the mechanism for increased Th1 cell infiltration.Publication without faculty affiliation
- Internalization of chemokine CXC receptor 3 in the CD3+ cells after ligand binding and endothelial transmigration suggest the mechanism for increased Th l synovial infiltration in rheumatoid arthritisPublication without faculty affiliation
- Periprosthetic osteolysis and its association with RANKL expression2007 | First Faculty of Medicine
- Immunolocalization of 3-nitrotyrosine in human synovium2006 | First Faculty of Medicine
- Immunolocalization of 3-nitrotyrosine in human synovium2006 | Faculty of Physical Education and Sport
- Increased expression of receptor activator of nuclear factor kappa B (RANK) and its ligand (RANKL) in rheumatoid arthritis synovial tissue2005 | First Faculty of Medicine
- Role of B lymphocytes in the pathogenesis of rheumatoid arthritis I. Subpopulations of synovial B lymphocytes differentially expressing IgM and IgG mRNA2005 | First Faculty of Medicine
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