Study programmes
Classes
- NeurobiologyMB150P36 | Faculty of Science
- NeurochemistryMB150P35 | Faculty of Science
- Experimental technics in neurosciencesMB150P43 | Faculty of Science
- Molecular PharmacologyMB150P47 | Faculty of Science
- Advances in Physiology and NeuroscienceMB150P54 | Faculty of Science
- Behavioral PharmacologyMB150P87 | Faculty of Science
- Stress physiologyMB150P96 | Faculty of Science
- Specialized seminar - Animal Physiology and Neurobiology +3MB150S02 | Faculty of Science
Publications
- Acute morphine affects the rat circadian clock via rhythms of phosphorylated ERK1/2 and GSK3 kinases and Per1 expression in the rat suprachiasmatic nucleus2015 | Faculty of Science
- Adenylyl Cyclase Signaling in the Developing Chick Heart: The Deranging Effect of Antiarrhythmic Drugs2014 | Faculty of Science, First Faculty of Medicine
- Chronic Hypoxia Enhances Expression and Activity of Mitochondrial Creatine Kinase and Hexokinase in the Rat Ventricular Myocardium2014 | Faculty of Science
- Adaptation to continuous and intermittent normobaric hypoxia is associated with similar alterations in the myocardial β-adrenergic signaling system2014 | Faculty of Science
- Transgenic rescue of defective Cd36 enhances myocardial adenylyl cyclase signaling in spontaneously hypertensive rats2013 | Faculty of Science, Central Library of Charles University
- Right-To-Left Ventricular Differences in the Expression of Mitochondrial Hexokinase and Phosphorylation of Akt2013 | Faculty of Science
- Global Changes in the Rat Heart Proteome Induced by Prolonged Morphine Treatment and Withdrawal2012 | Faculty of Science
- Identification of a Preassembled TRH Receptor-G(q/11) Protein Complex in HEK293 Cells2012 | Faculty of Science
- Identification and subcellular localization of molecular complexes of Gq/11α protein in HEK293 cells2012 | Faculty of Science
- Protein alterations induced by long-term agonist treatment of HEK293 cells expressing thyrotropin-releasing hormone receptor and G11alpha protein2010 | Faculty of Science
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