Abstract
Secondary dyslipidaemia is caused by a causal relationship to other disease or environmental factors. According to current knowledges, it is evident that the genetic background of the individual also plays an important role in this type of dyslipidaemia; this theory supports f.e. the individual's sensitivity to exogenous influences or lifestyle.
Typically, secondary dyslipidaemias are associated with different endocrinopathies (especially hypothyroidism), diabetes, nephropathies, hepatopathies, autoimmune diseases or alcohol abuse. A separate group consists of, not enough discussed, dyslipidaemias following drug administration, eg. diuretics, non-selective beta-blockers or immunosuppressants.
Physiologically, blood lipid values increase during pregnancy. In the case of finding of any dyslipidaemia, we must always exclude its possible secondary causes, which could lead to causal treatment.
Lipid-lowering therapy is indicated by persistent dyslipidaemia (after adequate causal treatment) in patients at high or very high cardiovascular risk. The choice of lipid-lowering therapy depends on the type of dyslipidaemia, or its etiology.